Allergic rhinitis (AR) is a typical type 2 immunoinflammatory disease, and the production of type 2 cytokines by type 2 helper T cells (Th2) and type 2 intrinsic lymphocytes (ILC2) is a central part of the type 2 immune response.ILC2 can be activated rapidly at the early stage of immune dysregulation, and synergistically with relatively late-activated Th2 cells, they can serve as a bridge between intrinsic and adaptive immunity. The synergy between Th2 cells and ILC2 constitutes a complex regulatory system in type 2 immune response. The aim of this paper is to understand the activation and inhibition mechanisms of ILC2, to sort out the interactions between innate ILC2 and adaptive Th2 cells, and to analyze the relationship between the two in type 2 immune response, which will help to further deepen the understanding of the pathogenesis of AR.